Neuro-Genix Global

Epigenome Modulation

Epigenome Modulation Process

The NG Epigenome Process may be used as a stand-alone therapeutic process or in conjunction with other complimentary or supplementary NG Processes. It is frequently combined with the NG Full Spectrum Brain Integration Processes and/or the NG Anti-Aging Process. Additional information on the NG Epigenome Process can also be found at

Epigenome Definition:

Cellular and physiological phenotypic (physical and biochemical characteristics) trait variations that are caused by external or environmental factors that switch genes on and off and affect how cells read genes instead of being caused by changes in the regular DNA sequence are the work of the Epigenome. The Epigenetic concept seeks to describe dynamic alterations in the transcriptional (building block) potential of the cells. Such alterations may or may not be heritable. Unlike genetics based on changes to the DNA sequence (the genotype), the changes in gene expression or cellular phenotype of epigenetics have other causes, thus use of the prefix epi- (Greek: επί- over, outside of, around). The term also refers to alterations or functionally relevant changes to the genome that do not involve a change in the nucleotide sequence. It is also important in an understanding of the epigenome that it is highly responsive to sensory data input and its subjective (e.g. personal or client/host/subject) perception and mental/ emotional interpretation. Our chemistry and neurology not only supports our existence it is also highly susceptible to it. In other words our personal attitudes, perceptions and unconscious programs have, as a rule, a much greater effect upon the epigenetic expression of our genes than any other factor.

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Interestingly, our attitudes and their expression—which serve as key inputs to the personal epigenetic expression of our DNA—are also highly affected by the same or similar aspects of our lineage, some of which can even be traced back to Adam and Eve and the roles and propensities of the man and the woman as they work out their joint contracts.

Epigenome Modulation Process Steps

1. Pre-Process Steps & Instructions

2. Optimum Process Flow Application Intelligence Request

3. Call to the Energetic Direct Organ/Circuit Activation/Modulation Utility (EDOCAM)

4. Call to the Celestial Field Orientation Attunement Utility (CFOAU)

5. Call to the Emotional State, Stress & Wave Form Clearing/Reprocess (ESSWFC/R) Utility

6. Clearing & Process Flow Instructions of Roots & Permissive Mind/Body States

7. Initial Client/Host Epigenome Session Scan

8. Additional Session Scans

9. Initial Epigenetic Discrepancies Session Scan

10. Nucleosome Properties Addressed

11. Critical Chromatin Structures Modulated

12. Optimization of Chromatin Structures

13. DNA Methylation & Related Modulation

14. Nutritional Support Factors

15. Histone Modification Modulation

16. Client/Host DNA/Histone Relationship Identification Scan

17. DNA/Histone Interaction History Registry Scan

18. Identification of Applicable Histone Modification Proteins and Related Methyl Groups

19. Histone Specific Base A & C Methylation Modification

20. Linker Histone H1 Nucleosome Regulation Supported

21. Histone H1 Role in Genomic Stabilization and Cancer Protection

22. Particular Histone (HDAC5) Positive Selective Effects

23. Noncoding DNA Factors Addressed

24. Epigenetic Mechanism’s Dual Nature

25. Accommodation of Two Transmission Routes

26. Epigenetic Signal Regulation of New Brain Cells

27. Transposable DNA Element (TE’s) Functions

28. Identification of Mutant Alleles & Base Pair Substitutions

29. Chromosomal Mutations Structure, Function and Inheritance

30. Feedback Loops Link Energy & its Information to RNA-Mediated Protein Folding

31. Virus-Driven Energy Theft & mRNA Degradation

32. RNA Interference (RNAi) & Chromosomal Rearrangements

33. RNA Interference (RNAi) & RNA Modulated System Stability

34. Nutrient Energy Dependent Endogenous DNA Expression

35. RNAi Gene Silencing & Repair

36. Energy-Dependent Cell Type Differentiation

37. Micro RNA (miRNA) Inhibition of DNA Expression

38. MicroRNA (miRNA), Chromatin & DNA Organization

39. Role of Small Interfering RNA (siRNA)

40. Untranslated Regions & miRNA’s/piRNA’s

41. MiRNA Processing/RNA & DNA Silencing

42. Down-Regulation of Genes Specifically

43. Up-Regulation of Genes Specifically

44. NMD Control & Modulation of DNA Expression

45. Placing & Monitoring Methyl Groups on DNA

46. Necessary Removal of Methyl Tags from DNA

47. RNA-Modification Therapeutic Application

48. Olfactory Effects on Gene Expression & Immune System Function

49. Entire Nervous System Interface

50. Role of Hsp90 Protein & Prions

51. FKPB5 Gene Trauma Risk Allele & Methylation

52. Common MTHFR Gene Mutation Compensation

53. Role of UHRF1 in Genetic Functions, Methylation & DNA Repair

54. Kinesin-3 motor Unc-104 Signaling Mediation of Mutant Synaptic Defects Modulated

55. Other Pernicious Gene Mutations & Genetic Disorders

56. Microbiome Effects upon DNA Transcription & Chronic Disease

57. Microbiome Effects on Client/Host Agency

58. Viral/Bacterial Onset & Residual Effects

59. Polycyclic Synthetic Effects Addressed & Modulated

60. Functional Mechanisms of the Glucocorticoid Receptor (GR)

61. GR-Dependent Gene Transcription

62. Genetic Algorithm Gene Encoding & mRNA Splice Variants

63. Types of Glucocorticoid Responsiveness (GR)

64. MicroRNA Involvement in Regulation of GR Expression

65. Periphery & CNS Involvement with GR & Depression

66. GR Environment Signaling & Function Homeostasis Maintenance

67. Specific Astrocyte/Microglial/TREM2 Genetic Issues

68. The Circadian Rhythm Clockwork Mechanism Optimized & Modulated

69. Maternal Care & Other Early Life Epigenetic Effects

70. Identification & Rectification of Infantile Trauma Induced DNA/Epigenetic Alterations

71. Sensory, Mental & Emotional Perception Affects & Effects

72. Energetic Signatures & Stealth Information Session Scan

73. Further DNA Damage Repair

74. Genetic/Epigenetic Optimum Life Purpose Performance Scan

75. Session Scan: Adam/Eve Covenant Considerations

76. Deep Transgenerational Epigenetic Effects

77. Call to Additional Modified/Extended Neuromodulation Pathways.

78. Client/Host Epigenetic History Rectification

79. Final Information Fault (IF) Translation for Clearing Purposes

80. Final Data Processing Instructions

81. Call to NG Behavioral & Habituation Change Path Process if Indicated

82. Final Process Objectives Prep

83. Final Clearing

For a more complete listing of the growing number of Neuro-Genix Processes see: